Literature Supports Trigger-Dispersion Theory :: Biology Essays Research Papers

Literature Supports Trigger-Dispersion Theory Having seen epileptic raptures and talking qualitatively rough the take in with people who have epilepsy, I made fivesome basic hypotheses about epilepsy. I collectively call these hypotheses my blow up dispersion theory of epilepsy. My five hypotheses atomic number 18 1 thither is an body politic in the brain where abnormal firing associated with seizures begins. I leave behind call this area the set off area. From the trigger area, abnormal firing spread outs to other areas of the brain compromising the function of the touch area. 2 There is a stimulus either external or internal which excites the trigger area. I will call this stimulus the trigger. The trigger can be very specific. 3 The first area touch on after the trigger area is sensorial.4 The abnormal firing spreads from the sensory area to an area for motor control. In this paper, I will go through the hypotheses of the trigger-dispersion theory and discuss litera ture that supports each hypothesis. Hypothesis 1- There is an area in the brain where abnormal firing associated with seizures begins. I will call this area the trigger area. From the trigger area, abnormal firing spreads to other areas of the brain compromising the function of the affected area. During a seizure, certain cells (a seizure focus) begin to abscond rapidly. In fact, nerve cells in the brain fire electrical impulses at a rate of up to four propagation higher than normal during a seizure (5) . This abnormal firing is spread this to other close cells. In the brain of an epileptic, there is not abundant inhibitory neurotransmitters to stop the spread of the abnormal firing (2) . In the 1800s, it was state by Jackson that epileptic seizures begin in isolated part of the body such as the thumb and from there spread to neighboring regions perhaps the arm and then to the rest of the body. He hypothesized that there were areas in cerebral cortex that controlled isolated m ovements and that the areas that were adjacent in the brain were anatomically adjacent as well. Therefore, a seizure began in one area and spread to the rest of the cortex. His hypothesis was later substantiated by Fritsch and Hittigs excitation experiments on motor cortex or area 4. It is a band of neural tissue on the cerebral cortex lying on precentral fissure. The bodys movements are mapped out on this band giving rise to the spreading mould that Jackson described during seizures (6) .